Maintenance of chondrocyte phenotype during expansion on PLLA microtopographies
نویسندگان
چکیده
منابع مشابه
Correction: Kinesin-1 promotes chondrocyte maintenance during skeletal morphogenesis
[This corrects the article DOI: 10.1371/journal.pgen.1006918.].
متن کاملKinesin-1 promotes chondrocyte maintenance during skeletal morphogenesis
During skeletal morphogenesis diverse mechanisms are used to support bone formation. This can be seen in the bones that require a cartilage template for their development. In mammals the cartilage template is removed, but in zebrafish the cartilage template persists and the bone mineralizes around the cartilage scaffold. Remodeling of unmineralized cartilage occurs via planar cell polarity (PCP...
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in the present study, the effect of e-64 at different concentrations (0.5, 1, 5 and 10 µm) added to (1) the ivm medium on oocyte nuclear maturation and developmental competence of ovine oocytes, and (2) to the ivc medium on embryonic development of ovine embryos were investigated.
Role of IFT88 in icariin-regulated maintenance of the chondrocyte phenotype
Maintenance of the chondrocyte phenotype is crucial for cartilage repair during tissue engineering. Intraflagellar transport protein 88 (IFT88) is an essential component of primary cilia, shuttling signals along the axoneme. The hypothesis of the present study was that IFT88 could exert an important role in icariin‑regulated maintenance of the chondrocyte phenotype. To this end, the effects of ...
متن کاملInfluence of species and anatomical location on chondrocyte expansion
BACKGROUND Bovine articular cartilage is often used to study chondrocytes in vitro. It is difficult to correlate in vitro studies using bovine chondrocytes with in vivo studies using other species such as rabbits and sheep. The aim of this investigation was to study the effect of species, anatomical location and exogenous growth factors on chondrocyte proliferation in vitro. METHODS Equine (E...
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ژورنال
عنوان ژورنال: Journal of Tissue Engineering
سال: 2018
ISSN: 2041-7314,2041-7314
DOI: 10.1177/2041731418789829